why season change affects ejaculation and orgasm frequency? on tempearture and light effects on LH, FSH, DHEA, testosterone, DHT, oxytocin, prolactin, prostaglandins, dopamine, acetylcholine and serotonin

Case Study: why season change affects ejaculation and orgasm frequency? on tempearture and light effects on LH, FSH, DHEA, testosterone, DHT, oxytocin, prolactin, prostaglandins, dopamine, acetylcholine and serotonin

Reader: 01/22/2008>Taoists believe that the season is very important to ejaculation frequency. Like they say its ok to ejaculate in the spring but dangerous to ejaculate too much in the winter. Is this true? And if it is, does it help if you live in a place where its always warm, even in the winter, like Florida?

Dr. Lin: 01/22/2008> If you live in up north when the average winter temperature is below 50 degrees F, it is true.
If you live in Florida, that is not true.
Our skin (including head skin and face) serves as a Peripheral Neuroendocrine organ. In the warm temperature, the Peripheral Neuroendocrine system can contribute about extra 10% of hormones and neurotransmitters for the entire body. Exposure to hot wealth can overheat your brain and nervous system. Light can also stimulate the skin to produce hormones and neurotransmitters, for example, Vitamin D. The visual/retinal circuit, as a part of the brain/nervous circuits, will also respond to light and object image to alternate release of hormones and neurotransmitters release. Our hormones LH, FSH, oxytocin, DHEA, testosterone and DHT, and neurotransmitters acetylcholine, dopamine and serotonin seasonally and positively respond to the environmental temperature and light. Therefore, in the cold area, there are more people experiencing winter depression. Sunbathing people are psychologically sexier and romantic. A warm temperature also stimulate tissues to release more prostaglandins, such as E1, E2 and E3, but the chilly temperature can only stimulate tissues or skin to produce prostaglandin E2 as a warning sign of the cold induced damage due to excessive conventional or radioactive heat loss. In a chilly temperature area, people will produce less nitric oxide and prostaglandins E1 and E, leading to arterial constriction and higher blood pressure. On the other hand, intensive heat can stimulate the skin, adrenal function and hypothalamus to produce excessive cortisol (hypercortisolism), dopamine, norepinephrine and epinephrine release for heat-induced stress, followed by heat stroke.

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