2-year antidepression medication results in low libido, erectile dysfunction and tight scrotum (testicular function disorders) for more more sexual orgasm. How antidepressants disable the dopamine-hypothalamus-pituitary-testicular (ovarian) axis for castration.

Case Study: 2-year antidepression medication results in low libido, erectile dysfunction and tight scrotum (testicular function disorders) for more more sexual orgasm. How antidepressants disable the dopamine-hypothalamus-pituitary-testicular (ovarian) axis for castration.

Reader: 8/20/2008> Age 26.
I have suffered from depression for many years. I have been on and off of anti-depressants. I am scared to take them for to long in fear of them causing medical problems. Over the past 2 years I have been unable to sustain a good erection. My interest in sex is very low and I rarely masturbate. Also my scrotum stays stays tight against my body even if I am hot. I am also trying to restore my foreskin through non-surgical means.
Thank you.

Dr. Lin: 8/20/2008>
Yes, the SSRIs antidepressant inhibits the reuptaking of serotonin in the nervous synapse by blocking serotonin transporter, so that the serotonin neurotransmitter can continue acting its effects on the postsynaptic neurons and possible outgrow the postsynaptic neurons (positive neurplasticity) under a long-term use . However, the SSRIs won't increase (actually decrease) the neurotransitter synthesis due to its intoxification effects on the liver Thus, SSRIs stimulates more serotonin neurons, but cuts down natural serotonin release from the nervous terminals. The SSRIs drug can also alter the temporal and spatial relationship between dopamine and serotionin signaling in the striatum which receives rich dopaminergic and more moderate serotonergic innervation. Under the high level of extracellular serotonin in the innervation synapses, elevated by exogenous application or by using antidepressants to inhibit the serotonin transporters, the extremely dense striatal Dopamine Transporters can uptake serotonin into dopamine terminals. This can destroy the dopamine nervous function responsible for pituitary oxytocin, LH and FSH release associated with sexual and testicular functions. A weak dopamine nervous function also results in a high pituitary prolactin release for disable sexual function.
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